Match your disease gene signature to drugs known to reverse it, or to drugs that produce a similar molecular effect.
Order cells along developmental or regenerative trajectories, using pseudotime to trace how states emerge, diverge, and transition.
Infer how your cell populations signal to one another, identifying the ligand-receptor interactions in your dataset.
Identify the biomarkers in your data that indicate a specific biological state, process, or treatment response.
Uncover the mechanism behind your signature with topology-aware impact analysis: perturbed pathways and the upstream regulators driving them.
Annotate single cells in your sample, automatically or manually (by cluster), to define which cell populations are present.