Infer how your cell populations signal to one another, identifying the ligand-receptor interactions in your dataset.
Annotate single cells in your sample, automatically or manually (by cluster), to define which cell populations are present.
Order cells along developmental or regenerative trajectories, using pseudotime to trace how states emerge, diverge, and transition.
Uncover the mechanism behind your signature with topology-aware impact analysis: perturbed pathways and the upstream regulators driving them.
Match your disease gene signature to drugs known to reverse it, or to drugs that produce a similar molecular effect.
Build interaction networks to identify hub genes, key drivers, upstream regulators, and predicted downstream effects.